International Journal of Pathology and Clinical Me 2009, 29(6) 483-486 DOI:     ISSN: 1673-2588 CN: 43-1458/R

Current Issue | Archive | Search                                                            [Print]   [Close]
REVIEW
Information and Service
This Article
Supporting info
PDF(1004KB)
[HTML]
Reference
Service and feedback
Email this article to a colleague
Add to Bookshelf
Add to Citation Manager
Cite This Article
Email Alert
Keywords
Helicobacter pylori;immune escape;CD4+CD25+ regulatory T cell;Toll-like receptors
Authors
PubMed

Role of CD4+CD25+regulatory T cells and TLRs in
immune escape of Helicobacter pylori

LIU Wei, XIE Yong

Institute of Digestive Disease, First Affiliated Hospital of Nanchang University; Key Laboratory
of Digestive Diseases of Jiangxi Province,Nanchang 330006, China

Abstract

After being infected by Helicobacter pylori, host will produce inflammation and immune response. However, the host can not completely clear Helicobacter pylori, one of the reasons is that Helicobacter pylori can escape host immune and form chronic persistent infection. The mechanisms underlying the immune escape of Helicobacter pylori are not clear yet. Currently, the studies are mainly focusing on the role of CD4+CD25+regulatory T cells and TLRs in  immune escape of Helicobacter pylori.

Keywords Helicobacter pylori;immune escape;CD4+CD25+ regulatory T cell;Toll-like receptors  
Received 2009-08-10 Revised 2009-09-03 Online:  
DOI:
Fund:
Corresponding Authors:
Email: xieyong_med@163.com
About author:

References:
Similar articles

Copyright by International Journal of Pathology and Clinical Me